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Protocols: Metabolic Syndrome

General Medicine, Internists, Endocrinologists

Applying the WHO’s definition of metabolic syndrome, nearly 10% of people with normal glucose tolerance, 40% of people with glucose intolerance, and 90% of patients with type 2 diabetes could have metabolic syndrome.

Metabolic syndrome (also known as syndrome X, plurimetabolic syndrome, insulin resistance syndrome, Reaven’s syndrome, or CHAOS in Australia) is the cluster of conditions or risk factors that occur together in an individual that increase the person's risk of cardiovascular diseases or diabetes mellitus.

In the United States, according to the definition of metabolic syndrome provided by the NCEP (National Cholesterol Education Program-Adult Treatment Panel III), approximately 25% of the population over 20 years of age suffers from metabolic syndrome, which is the point at which an individual suffers from central obesity (concentrated on the abdomen) and insulin resistance.at the same time.

Given the significance of the relationship between insulin resistance and metabolic syndrome, an explanation of the physiopathalogical mechanisms is in order. The theory on metabolism holds that the compensatory hyperinsulinemia that results  from insulin resistance is the factor responsible for hypertension, type 2 diabetes, dyslipidemia, obesity, endothelial dysfunction and atherosclerosis, through several mechanisms.

The cause of metabolic syndrome is unknown. Its physiopathology is extremely complex and is only partially understood. The majority of patients are of considerably advanced age, obese, sedentary, and have a certain level of insulin resistance. Insulin resistance plays a central role in the development of this syndrome.

Hyperinsulinemia, or the elevated concentration of insulin in blood plasma, turns out to be an independent risk factor for the development of ischemic heart disease, contributes to the early onset and subsequent progression of diabetes and contributes to the development of a number of other associated pathologies that translate into cardiovascular risk factors.

What we do know is that this disease is associated with high levels of oxidative stress, which leads to the progressive deterioration of tissues.
Antioxidant support is considered to be of vital importance in mitigating the effects of the disease.

Antioxidants are chemical substances that inhibit or delay the oxidation of various substances, mainly fatty acids, whose reactions are produced in food as well as in the human body, where they can trigger significant physiological changes that can in turn cause different diseases. Another function of antioxidants is to facilitate the physiological use of oxygen by cell mitochondria, helping to reduce the effects of oxidative stress and lack of oxygen and forming complexes that mitigate the reactions that produce oxygen radicals, also known as free radicals.

Free radicals are unstable, high-energy molecules with missing electrons in their outer orbits which tend to react with other compounds. Thus, antioxidants play a fundamental role in the prevention of non-infectious chronic diseases. Antioxidants are classified as either enzymatic (also known as endogenous) or non-enzymatic (also known as exogenous), and they can be active in both the intracellular and extracellular space.

 

Universal Protocol (Support for Pancreas, Cardiovascular System, and Anti-oxidant Systems as Cellular Revitalization Support)

Cellorgane Multi-complex No. 10 Pancreas Oral

  • Cellorgane Multi-complex No. 12 tablets. Take 2 tablets in the morning and 2 tablets at night (both times on an empty stomach). Treatment should be taken continuously for at least 3 months, and then re-evaluate.

Revercell

  • To be taken concurrently with the previous treatment. One white capsule in the morning, one white/red capsule in the afternoon, and one red capsule at night before meals, contin-uously for six months, and then re-evaluate.

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